Article Information
PubMed
Published By
History
- Received June 19, 2018
- Accepted June 27, 2018
- Published online July 24, 2018.
Copyright & Usage
Copyright © 2018 Kühnl et al. This is an open-access article distributed under the terms of the Creative Commons Attribution 4.0 International license.
Editors and / or Reviewers
- Jean Gruenberg, Invited Editor, University of Geneva
- Scott D. Emr, Editor, Cornell University
Author Information
- Alexander Kühnla,c,d,
- Agnes Musiola,c,d,
- Nicole Heitziga,c,d,
- Danielle E. Johnsone,
- Christina Ehrhardtb,c,d,
- Thomas Grewalf,
- Volker Gerkea,c,d,
- Stephan Ludwigb,c,d,
- Ursula Reschera,c,d
- aInstitute of Medical Biochemistry, Center for Molecular Biology of Inflammation, University of Muenster, Muenster, Germany
- bInstitute of Virology, Center for Molecular Biology of Inflammation, University of Muenster, Muenster, Germany
- cInterdisciplinary Centre for Clinical Research, University of Muenster, Muenster, Germany
- dCluster of Excellence “Cells in Motion,” University of Muenster, Muenster, Germany
- eProgram in Cell Biology, Hospital for Sick Children, Toronto, Ontario, Canada
- fFaculty of Pharmacy A15, University of Sydney, Sydney, NSW, Australia
- Address correspondence to Ursula Rescher, rescher{at}uni-muenster.de.
Citation Kühnl A, Musiol A, Heitzig N, Johnson DE, Ehrhardt C, Grewal T, Gerke V, Ludwig S, Rescher U. 2018. Late endosomal/lysosomal cholesterol accumulation is a host cell-protective mechanism inhibiting endosomal escape of influenza A virus. mBio 9:e01345-18. https://doi.org/10.1128/mBio.01345-18.