Research Article

Zika Virus Persistently Infects and Is Basolaterally Released from Primary Human Brain Microvascular Endothelial Cells

Megan C. Mladinich, John Schwedes, Erich R. Mackow
Glen Nemerow, Editor
DOI: 10.1128/mBio.00952-17

Article Information

DOI 
https://doi.org/10.1128/mBio.00952-17
PubMed 
28698279
Published By 
American Society for Microbiology
History 
  • Received June 5, 2017
  • Accepted June 8, 2017
  • Published online July 11, 2017.
Copyright & Usage 
Copyright © 2017 Mladinich et al. This is an open-access article distributed under the terms of the Creative Commons Attribution 4.0 International license .
Editors and / or Reviewers 
  • Glen Nemerow, Editor, Scripps Research Institute
  • Christopher Basler, Solicited external reviewer, Georgia State University
  • Sujan Shresta, Solicited external reviewer, La Jolla Institute for Allergy and Immunology

Author Information

  1. Megan C. Mladinicha,c,
  2. John Schwedesb,
  3. Erich R. Mackowa,c
  1. a Department of Molecular Genetics and Microbiology, Stony Brook University, Stony Brook, New York, USA
  2. b Genomics Core Facility, Stony Brook University, Stony Brook, New York, USA
  3. c Molecular and Cell Biology Program, Stony Brook University, Stony Brook, New York, USA
  • Address correspondence to Erich R. Mackow, Erich.Mackow{at}stonybrook.edu.

  • Citation Mladinich MC, Schwedes J, Mackow ER. 2017. Zika virus persistently infects and is basolaterally released from primary human brain microvascular endothelial cells. mBio 8:e00952-17. https://doi.org/10.1128/mBio.00952-17 .

  1. This article is a direct contribution from a Fellow of the American Academy of Microbiology.

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KEYWORDS

brain
endothelial cells
microvessels
Virus Release
virus replication
Zika virus
basolateral release
chemokine CCL5
IFN-β regulation
ISG15 induction
persistent infection
Zika virus
cell survival
human brain endothelial cells
innate immune regulation
transcriptome analysis

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